In a recently published study, researchers revealed that NSD2 overexpression in t(4;14) multiple myeloma (MM) redirects S-adenosylmethionine (SAM) toward excessive methylation of the epigenome, specifically histone H3K36 dimethylation.
NSD2 Overexpression in t(4;14) Multiple Myeloma Identifies AK2 as a New Therapeutic Target
Kate Bondaruk2024-10-11T18:50:04+00:00October 11th, 2024|multiple myeloma|Comments Off on NSD2 Overexpression in t(4;14) Multiple Myeloma Identifies AK2 as a New Therapeutic Target